In a simulated motoneuron, we studied the effects of tonic coactivation of glutamatergic (NMDA-type) synapses covering the somato-dendritic membrane and of GABA-ergic synapses located on the axon hillock. As in the prototypes, NMDA activation caused oscillatory plateau potentials with the bursts of action potentials (AP). Plateau depolarizations spreading from the soma inactivated <$E roman Na sup +> channels and reduced the number of AP in the axon compared with that in the soma. As GABA activation increased, interplateau intervals also increased, while the plateau duration and number of AP per burst decreased.

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